The emerging evidence that changes in the skin microbiota, known as dysbiosis, can promote inflammation has attracted scientific interest in the “microbial therapy” or the use of live or inactivated microorganisms and substances produced to rebalance the physiological skin microbiota aiming to restore the correct functioning of the skin barrier and therefore the return to a non-pathological condition.

This research intends to investigate the role of microorganisms generally associated with inflammatory skin diseases: atopic dermatitis, acne, psoriasis and pressure sores by analyzing their interference with the growth of other diners to investigate possible strategies capable of preventing the onset and/or the chronicity of these pathologies.

Several tests have been conducted both on pathogenic microorganisms (bacteria and fungi) Staphylococcus aureus and Candida albicans and on non-pathogenic ones: Staphylococcus epidermidis and Roseomonas mucosa.